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A scientific proof in the
field of Medical Sciences.
Doctoral dissertation
to be presented by permission
of the Faculty of Medicine
of the University of Kuopio,
for public examination
in Auditorium L3, Canthia building,
University of Kuopio,
on Friday, August 15, 1997
at 12.00 a.m.
By:
Pauli Kilpeläinen |
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I
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Reliability
of the moiré method in study of tooth and palatal morphology.
Kilpeläinen P, Laine T, Väyrynen M.
Published in Proc Finn Dent Soc, 1990; 86:89-97.
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II
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Palatal morphology
and type of clefting.
Kilpeläinen PVJ, Laine-Alava MT, Lammi S.
Published
in
Cleft Palate-Craniofac J, 1996; 33:477-482.
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III
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Palatal asymmetry
in cleft palate subjects.
Kilpeläinen PVJ, Laine-Alava MT.
Published
in Cleft
Palate-Craniofac J, 1996; 33:483-488
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IV
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Upper airway
function and orofacial morphology in cleft palate subjects.
Kilpeläinen PVJ, Laine-Alava MT, Kuijpers-Jagtman AM.
Submitted to Cleft Palate-Craniofac J.
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V
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Velopharyngeal
function in relation to the type of clefting and palatal morphology.
Laine-Alava MT, Kilpeläinen PVJ, Kuijpers-Jagtman AM.
Submitted
to Cleft
Palate-Craniofac J.
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The purpose of
this investigation was to study the palatal morphology and oral-nasal
function among cleft palate patients with different types of clefts. The
sample consisted of 95 subjects with various types of clefts, all including
cleft palate and 68 noncleft individuals.
The moir‚
technique was modified and applied to evaluate palatal morphology. Its
reliability was tested and proved to be good. Craniofacial morphology
was evaluated from lateral cephalometric films. For measuring rest breathing
and velopharyngeal function during speech production, the pressure-flow
technique was utilized.
Compared to noncleft
individuals, the cleft palate subjects had a smaller palatal height, depth,
and width, a more asymmetrical location of the upper first molars, a more
deviant shape of the palate. Multiple regression showed that the different
cleft types had a different effect on palatal dimensions and symmetry,
especially those cleft types that involved the secondary palate had a
decreased palatal height. The cleft type affected both the anteroposterior
and transverse position of the highest point of the palatal vault.
During rest breathing
cleft palate patients had a larger differential pressure and nasal resistance,
and a smaller cross-sectional area and nasal flow rate than noncleft individuals.
The individuals in which the cleft was located in the anterior part of
the palate had a decreased nasal cross-sectional area and airflow rate
but the nasal resistance was not affected. Midfacial morphology, especially
vertical facial proportions were related to decrease in nasal cross-sectional
area and increase in the nasal resistance while the effect of palatal
morphology on rest breathing was small. Those cleft individuals, who had
adequate upper airway size, were able to breathe normally.
During speech production
nasal airflow rate and velopharyngeal orifice area were increased in cleft
palate patients, but oral-nasal differential pressure was not different
from the control group. In the cleft types in which the cleft was located
in the posterior part of the palate, velar function was affected. Craniofacial
and pharyngeal features, like the length of the palate, depth of the nasopharynx
and those features reflecting vertical facial discrepancies were associated
with velar function. Hypodontia and the configuration of the posterior
palate had also some effect on the velar function, but this was minor
and improved with age.
Different cleft
types seem to have a different kind of effect on palatal morphology and
oral-nasal functions and therefore the cleft type should be taken into
account in the treatment planning of the cleft palate patients. The existence
of a cleft as such without decreased upper airway size does not lead to
nasal impairment. The morphology of the palate affects more velar function
than rest breathing. The point that the differential pressure did not
differ between cleft and noncleft subjects supports the pressure maintenance
theory during speech production. Individuals with severe clefts compansate
their velopharyngeal insufficiency by increased nasal airflow rate sustaining
adequate pressure for speech.
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